Archive for October, 2014

11/07/14: Promoting Emotional Well-Being and Preventing Suicide Among Asian/Asian-American University Students

This conference is designed to help clinicians, health care professionals, university staff and community

advocates who provide direct service to Asian/Asian American students:

  • Identify contributing factors to suicide and discuss potential barriers to access
  • Describe emerging ideas for culturally sensitive outreach and promoting emotional well-being
  • Demonstrate knowledge of current practices and discuss future directions related to service delivery and
  • clinical intervention on various local campuses.
  • Create collaborations, share community resources, and expand supportive networks

For questions, please call the Caltech Counseling Center at 626-395-8831 or email

INTRODUCING Eastside Psychological Services, Inc.

Eastside Psychological Services, Inc.

 “Committed to the Welfare of Children and the Betterment of Families”

Eastside Psychological Services, Inc. has been  providing mental health  services to the children and their family members in our community since 1992.  We are a provider for The Child Abuse Prevention and Treatment Services (CAPTS) Contract.  We employ research based treatments that are individualized and comprehensive for our clients.

Services are provided in English, Spanish, Chinese, Italian, and Vietnamese, throughout Los Angeles, Orange, San Bernardino, and Riverside Counties.

We offer After-Hours/Weekends In-Office, In-Home, and In-School treatment.

Type of Services:

  • Individual Therapy
  • Family/Couples Therapy
  • PCIT/Floor Play
  • Bonding/Attachment Assessment
  • Psychological Evaluation and Testing/Report


111 S. Garfield Avenue, Suite 101
Montebello, CA 90640
(323) 890-0180 (Office)
(323) 890-1167 (Fax)

Other Locations:

  • San Bernardino County
  • Riverside County
  • Orange County

(Toll-Free) (800) 630-4808

Eastside Referral Form

Rapid agent restores pleasure-seeking ahead of other antidepressant action | Depression deconstructed — NIH study


For Immediate Release: Friday, October 17, 2014

Rapid agent restores pleasure-seeking ahead of other antidepressant action

Depression deconstructed — NIH study

A drug being studied as a fast-acting mood-lifter restored pleasure-seeking behavior independent of — and ahead of — its other antidepressant effects, in a National Institutes of Health trial. Within 40 minutes after a single infusion of ketamine, treatment-resistant depressed bipolar disorderpatients experienced a reversal of a key symptom — loss of interest in pleasurable activities — which lasted up to 14 days. Brain scans traced the agent’s action to boosted activity in areas at the front and deep in the right hemisphere of the brain.

PET scans revealed that ketamine rapidly restored bipolar depressed patients’ ability to anticipate pleasurable experiences by boosting activity in the dorsal anterior cingulate cortex (yellow) and related circuitry. Picture shows PET scan data superimposed on anatomical MRI image.
“Our findings help to deconstruct what has traditionally been lumped together as depression,” explained Carlos Zarate, M.D., of the NIH’s National Institute of Mental Health. “We break out a component that responds uniquely to a treatment that works through different brain systems than conventional antidepressants — and link that response to different circuitry than other depression symptoms.”

This approach is consistent with the NIMH’s Research Domain Criteria project, which calls for the study of functions – such as the ability to seek out and experience rewards – and their related brain systems that may identify subgroups of patients in one or multiple disorder categories. Zarate and colleagues reported on their findings Oct. 14, 2014 in the journal Translational Psychiatry.

Although it’s considered one of two cardinal symptoms of both depression and bipolar disorder, effective treatments have been lacking for loss of the ability to look forward to pleasurable activities, or anhedonia. Long used as an anesthetic and sometimes club drug, ketamine and its mechanism-of-action have lately been the focus of research into a potential new class of rapid-acting antidepressants that can lift mood within hours instead of weeks.

Based on their previous studies, NIMH researchers expected ketamine’s therapeutic action against anhedonia would be traceable — like that for other depression symptoms — to effects on a mid-brain area linked to reward-seeking and that it would follow a similar pattern and time course.To find out, the researchers infused the drug or a placebo into 36 patients in the depressive phase of bipolar disorder. They then detected any resultant mood changes using rating scales for anhedonia and depression. By isolating scores on anhedonia items from scores on other depression symptom items, the researchers discovered that ketamine was triggering a strong anti-anhedonia effect sooner — and independent of — the other effects.

Levels of anhedonia plummeted within 40 minutes in patients who received ketamine, compared with those who received placebo — and the effect was still detectable in some patients two weeks later. Other depressive symptoms improved within 2 hours. The anti-anhedonic effect remained significant even in the absence of other antidepressant effects, suggesting a unique role for the drug.

Next, the researchers scanned a subset of the ketamine-infused patients, using positron emission tomography (PET), which shows what parts of the brain are active by tracing the destinations of radioactively-tagged glucose — the brain’s fuel. The scans showed that ketamine jump-started activity not in the middle brain area they had expected, but rather in the dorsal (upper) anterior cingulate cortex, near the front middle of the brain and putamen, deep in the right hemisphere.

Boosted activity in these areas may reflect increased motivation towards or ability to anticipate pleasurable experiences, according to the researchers. Depressed patients typically experience problems imagining positive, rewarding experiences — which would be consistent with impaired functioning of this dorsal anterior cingulate cortex circuitry, they said. However, confirmation of these imaging findings must await results of a similar NIMH ketamine trial nearing completion in patients with unipolar major depression.

Other evidence suggests that ketamine’s action in this circuitry is mediated by its effects on the brain’s major excitatory neurotransmitter, glutamate, and downstream effects on a key reward-related chemical messenger, dopamine. The findings add to mounting evidence in support of the antidepressant efficacy of targeting this neurochemical pathway. Ongoing research is exploring, for example, potentially more practical delivery methods for ketamine and related experimental antidepressants, such as a nasal spray.

However, Ketamine is not approved by the U.S. Food and Drug Administration as a treatment for depression. It is mostly used in veterinary practice, and abuse can lead to hallucinations, delirium and amnesia.

About the National Institute of Mental Health (NIMH): The mission of the NIMH is to transform the understanding and treatment of mental illnesses through basic and clinical research, paving the way for prevention, recovery and cure. For more information, visit

About the National Institutes of Health (NIH): NIH, the nation’s medical research agency, includes 27 Institutes and Centers and is a component of the U.S. Department of Health and Human Services. NIH is the primary federal agency conducting and supporting basic, clinical, and translational medical research, and is investigating the causes, treatments, and cures for both common and rare diseases. For more information about NIH and its programs, visit

NIH…Turning Discovery Into Health®


Anti-anhedonic effect of ketamine and its neural correlates in treatment-resistant bipolar depression. Lally N, Nugent AC, Luckenbaugh DA, Ameli R, Roiser JP, Zarate CA. Transl Psychiatry. 2014 Oct 14;4:e469. doi: 10.1038/tp.2014.105. PMID: 25313512

10/12/14: memorial service for Aniya Parker (trans woman killed in east Hollywood)

Please support our local trans community

Los Angeles County HIV Drug & Alcohol Task Force

A Memorial Service is being held for Aniya Parker at the Unity Fellowship Church, 9608 S. Figueroa, this Sunday October 12 at 7:30 pm.

Please plan to get there early as the Church may be full. There will be an opportunity for community members to speak for 5 minutes max, the Reverend said he is very strict about the time.

There will be press covering the service.  I haven’t heard yet if Aniya’s family will be a part of the service.

The Church and members of the black community are taking the lead.  The Anti-Violence Project at the Center is playing a supportive role.

I will send further updates.

There is also a campaign to raise money for Aniya’s funeral expenses. www.gofundme/aniyaparker

FOR DETAILS ABOUT THE CRIME: (the coverage goes from atrocious to better as time progresses)

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meeting reminder and agenda for October’s meeting

This is a reminder that our monthly meeting will take place on Wednesday, October 8, 2014 at 9:30 am at the Village [1125 N. McCadden Place, LA, 90038.]

Here is the agenda:

REMINDER about today’s HIV Housing Resource Fair!

Reminder that today there is a housing fair for both providers and community members living with HIV.

Los Angeles County HIV Drug & Alcohol Task Force


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